Researchers tested experimental PCAI compounds against pancreatic cancer cells and found they had powerful anticancer effects. One leading compound blocked more than 90% of cancer cell migration, suggesting it could help prevent the spread of tumors. Rather than suppressing cancer signaling, the treatment hyperactivated key pathways until the cells essentially self-destructed.
Firstly, I want to second that this explanation was very good. Second, can you explain why funding would not be available to study why this drug is effective? To me it would seem that this could be a master key that unlocks treatment options for other types of cancers
“hey what if we tried to make cancer grow faster” is not a great pitch
they got funding because it wasn’t what they were trying to do, but it did ended up being the case, but it worked anyway so, so far, all is good. but don’t expect that it will become a new drug, because 90% of clinical trials fail, and here maybe there is some other less malformed cancer line that will grow faster after administration of that drug, which is sort of what was could be expected now that we know mechanism